Terconazole is a synthetic antifungal agent that belongs to the class of imidazole derivatives. It is commonly used to treat vaginal yeast infections caused by Candida albicans. Terconazole is available in various forms, including creams, suppositories, and tablets.
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(E)-N-[2-(2-methylindol-1-yl)ethyl]-3-(3,4,5-trimethoxyphenyl)prop-2-enamide, also known as MIPEP, is a synthetic compound that has gained attention in the scientific community due to its potential therapeutic and environmental applications. MIPEP is a member of the propenamide family and has a molecular weight of 416.5 g/mol.
Novel inhibitor of tissue transglutaminase (TG2) and fibronectin (FN) protein-protein interaction TG53 is an inhibitor of tissue transglutaminase (TG2) and fibronectin (FN) protein-protein interaction.
5-(Pyridin-4-yl)-1H-indazole is a heterocyclic compound that has gained significant attention in recent years due to its potential therapeutic and industrial applications. This paper aims to provide an overview of the synthesis, chemical structure, biological activity, and applications of 5-(Pyridin-4-yl)-1H-indazole, as well as future perspectives and challenges.
Prostaglandin E2 (PGE2;) evokes distinct responses through four different ‘E prostanoid’ (EP) receptors. EP2 is a G protein-coupled receptor that has diverse roles, including those in cancer, inflammation, and neuroprotection. TG6-129 is an antagonist of the EP2 receptor, suppressing PGE2-induced elevation of cAMP in cells expressing EP2 with an IC50 value of 1.6 µM. It is without effect on EP4, DP1, IP, and β2-adrenergic receptors. TG6-129 reduces the expression of COX-2, IL-1β, IL-12, IL-23, IL-6, and TNF-α induced by the EP2-selective agonist butaprost in P388D1 macrophages. It has low cell cytotoxicity (CC50 = 326 µM), prolonged plasma half-life, and does not cross the blood-brain barrier. TG6-129 is an antagonist of the EP2 receptor, suppressing PGE2-induced elevation of cAMP in cells expressing EP2 with an IC50 value of 1.6 µM. It is without effect on EP4, DP1, IP, and β2-adrenergic receptors. TG6-129 reduces the expression of COX-2, IL-1β, IL-12, IL-23, IL-6, and TNF-α induced by the EP2-selective agonist butaprost in P388D1 macrophages.